Fig. 1.  Representative traces depicting the effect of time (A ), propofol (10 μm; B ), propofol in the presence of the specific protein kinase C epsilon (PKCϵ) inhibitor peptide (ϵV1-2; 0.5 μm; C ), and the specific PKCϵ activator peptide (ψϵRACK; 0.5 μm; D ) after capsaicin-induced (100 nm) desensitization on restoration of transient receptor potential vanilloid receptor type 1 sensitivity in mouse dorsal root ganglion neurons. Summarized data for A–D , including 1 and 5 μm propofol, are depicted in E . Data are expressed as a percentage of the response to the final application of capsaicin (Cap) in the untreated control (% of Control). *P < 0.05 compared with final Cap in the untreated control. # P < 0.05 compared with propofol (Pro) plus Cap. n = DRG neurons from six different mice.