Fig. 4. Carboetomidate modulation of human γ-aminobutyric acid (GABA) type A receptor function. (A ) Representative traces showing reversible enhancement by 10 μm carboetomidate of currents mediated by wild-type receptors when evoked by EC^5–10γ-aminobutyric acid. (B ) Representative traces showing minimal enhancement by 10 μm carboetomidate of currents mediated by etomidate-insensitive mutant receptors when evoked by EC^5–10γ-aminobutyric acid.