Fig. 8.
Ischemia–reperfusion with spinal cord stimulation increases γ-aminobutyric acid type A (GABAA) but not γ-aminobutyric acid type B (GABAB) receptor expression in dorsal horn. (A and B) The expression of GABAAα and GABAAβ was significantly greater in the dorsal horn of the ischemia–reperfusion plus spinal cord stimulation group than the ischemia–reperfusion and control groups. (C and D) There was no significant difference in the expression of GABAB receptor 1 and GABAB receptor 2 among the three groups. (E to H) Representative images of each membrane used for analysis. Glyceraldehyde-3-phosphate dehydrogenase was used as a loading control. (A) *P = 0.004 versus control, (B) *P = 0.048 versus control, and *P = 0.031 versus ischemia–reperfusion. Control (n = 5). Ischemia–reperfusion (n = 5). Ischemia–reperfusion plus spinal cord stimulation (n = 5).

Ischemia–reperfusion with spinal cord stimulation increases γ-aminobutyric acid type A (GABAA) but not γ-aminobutyric acid type B (GABAB) receptor expression in dorsal horn. (A and B) The expression of GABAAα and GABAAβ was significantly greater in the dorsal horn of the ischemia–reperfusion plus spinal cord stimulation group than the ischemia–reperfusion and control groups. (C and D) There was no significant difference in the expression of GABAB receptor 1 and GABAB receptor 2 among the three groups. (E to H) Representative images of each membrane used for analysis. Glyceraldehyde-3-phosphate dehydrogenase was used as a loading control. (A) *P = 0.004 versus control, (B) *P = 0.048 versus control, and *P = 0.031 versus ischemia–reperfusion. Control (n = 5). Ischemia–reperfusion (n = 5). Ischemia–reperfusion plus spinal cord stimulation (n = 5).

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