Fig. 7. θ-Band power during recovery from etomidate anesthesia in the hippocampus (A ) and prefrontal cortex (B ) of wild-type and β3(N265M) mutant mice. Bar plots  indicate θ power 40–60 min and 80–100 min after etomidate injection. The first time window matches the period shortly after righting reflexes reappeared in wild-type animals. Therefore, it coincides with an early phase of emergence from anesthesia. During this period, etomidate caused a significant increase in θ power in the hippocampus but not in the prefronatal cortex. This action is sensitive to the β3(N265M) mutation. It was absent in sham-injected animals of both genotypes (**P < 0.01). RoRR = recovery of righting reflexes.

Fig. 7. θ-Band power during recovery from etomidate anesthesia in the hippocampus (A ) and prefrontal cortex (B ) of wild-type and β3(N265M) mutant mice. Bar plots  indicate θ power 40–60 min and 80–100 min after etomidate injection. The first time window matches the period shortly after righting reflexes reappeared in wild-type animals. Therefore, it coincides with an early phase of emergence from anesthesia. During this period, etomidate caused a significant increase in θ power in the hippocampus but not in the prefronatal cortex. This action is sensitive to the β3(N265M) mutation. It was absent in sham-injected animals of both genotypes (**P < 0.01). RoRR = recovery of righting reflexes.

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