Figure 1. Recording of surface electrocardiographic lead V6in a malignant hyperthermia (MH)-susceptible (MHS) pig during baseline recordings (t = 0) and after 10, 20, 40, and 90 min of halothane exposure (1% inspired) in addition to succinylcholine (3 mg *symbol* kg sup -1 intravenously 15 min after the start of halothane exposure). This recording is not entirely representative of the MHS group because arrhythmias occurred intermittently during each phase of MH. However, wide QRS complexes and elevated T waves occurred in the final phase of MH.

Figure 1. Recording of surface electrocardiographic lead V6in a malignant hyperthermia (MH)-susceptible (MHS) pig during baseline recordings (t = 0) and after 10, 20, 40, and 90 min of halothane exposure (1% inspired) in addition to succinylcholine (3 mg *symbol* kg sup -1 intravenously 15 min after the start of halothane exposure). This recording is not entirely representative of the MHS group because arrhythmias occurred intermittently during each phase of MH. However, wide QRS complexes and elevated T waves occurred in the final phase of MH.

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